This article written by Dr. William Brady, an ART provider, details the importance and effects of soft tissue injuries and adhesion formation following such injuries.
Hypoxic Fibrous Adhesion Production: Chemical Pathway and Clinical Importance
Physicians need reliable terms that describe normal and pathological conditions of lumbar discs. Terms that can be interpreted accurately,consistently, and with reliable precision are particularly important for communicating impressions gained from imaging for clinical diagnostic and therapeutic decision making. The following paper focuses on this issue and has become the standard in the field of spine medicine.
Published in Spine Volume 26(5) 1 March 2001
By Warren Hammer, MS, DC, DABCO
This article was origionally published in Dynamic Chiropractic in the April 22, 2002 Vol. 20, Issue 9
Tendonitis is a misnomer. With microtraumatic overuse injury, we are dealing most of the time with tendinosis. While the stages of pathology of the macrotraumatic inflammatory response are well-known, the tissue pathology of tendinosis needs more clarity.
In conditions such as tennis elbow, and overuse injuries in general, there is a fibroblastic and vascular response (tendinosis), rather than an immune blood-cell inflammatory response.1 In a normal tendon, collagen fibrils are embedded in a matrix of proteoglycans, glycosaminoglycans and water, surrounded by a loose connective tissue containing visceral and parietal layers. Vascularized tendons (de Quervain, etc.) are surrounded by epitenon with capillaries and penetrating vessels.2
By Warren Hammer, MS, DC, DABCO
This article origionally was published in Dynamic Chiropractic November 6, 1992 Vol. 10, Issue 23
It is often assumed that if there is pain, there must be inflammation. But on second thought, we realize that a myofascial trigger point or even fibrositis (which implies inflammation) may have a neurogenic origin and not be inflamed.
This article is not intended to encourage you to vaccinate or not to vaccinate against the flu, but rather to provide you with some basic information so you can decide for yourself.
The following article was written by Holly J Benjamin, MD, FACSM, FAAP and Igor Boyarsky, DO, was published on medscape on Jan 5, 2009 and is listed here as a reference detailing Little League Elbow.
The normal response to injury in the body is inflammation and the formation of scar tissue. The following PDF reviews the stages of healing from acute injury through recovery.
Sacroiliac Joint Pain: A Comprehensive Review of Anatomy, Diagnosis, and Treatment
Steven P. Cohen, MD
Current Research on Muscle Hyperplasia and Exercise
Each skeletal muscle fiber is a syncytium and develops by the fusion of many myoblasts. Myoblasts proliferate extensively, but once they have fused, they can no longer divide. Fusion generally follows the onset of myoblast differentiation, in which many genes encoding muscle-specific proteins are switched on coordinately. Some myoblasts persist in a quiescent state as satellite cells in adult muscle; when a muscle is damaged, these cells are reactivated to proliferate and to fuse to replace the muscle cells that have been lost. Muscle bulk is regulated homeostatically by a negative-feedback mechanism, in which existing muscle secretes myostatin, which inhibits further muscle growth
The soft tissues of the body(muscles, tendons, and ligaments) control the movement of the joints. Muscle imbalance due to contracted soft tissues that results from repetitive use or minor sports related trauma can accumulate over months and years. Active Release Techniques® is a medically patented technique for soft tissue management that can detect and alleviate adhesions and scar tissue that is restricting muscle and joint function. When proper soft tissue treatment is paired correctly with corrective rehabilitative exercise, many of the overuse chronic and nagging injuries can be completely resolved and allow a full return to activity and sport.
The female athlete triad put forth by the ACSM in 1997 was defined as a triad of disordered eating, amenorrhea, and osteoporosis affecting female athletes. Research on the female athlete triad for the past decade or so has culminated in an updated definition published by ACSM. The 2007 ACSM positional stand looks at each disorder as it exists on a continuous spectrum instead of a serious pathologic endpoint.
NOTE: The Female Athlete Triad has now become the Female Athlete Tetrad. Medical researchers have added cardiovascular disease after discovering that many women with the other three health issues often have a dysfunction with thier blood vessels, making them more likely to develop cardiovascular diseases.
For more Info go to http://www.femaleathletetriad.org/
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